Possible contributions of a novel form of synaptic plasticity in Aplysia to reward, memory, and their dysfunctions in mammalian brain

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Figure 3.
Figure 3.

Novel synaptic (A) and intrinsic (B) mechanisms in hippocampus and entorhinal cortex (EC) that could be involved in reference memory, working memory, and their dysfunction in Alzheimer’s disease. (TRP) Transient receptor potential.

This Article

  1. Learn. Mem. 20: 580-591

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