Possible contributions of a novel form of synaptic plasticity in Aplysia to reward, memory, and their dysfunctions in mammalian brain

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Figure 2.
Figure 2.

Novel synaptic (A) and circuit (B) mechanisms in ventral tegmental area (VTA) and nucleus accumbens (NAc) that could be involved in reward and its dysfunction in addiction. (PFC) Prefrontal cortex, (PTg) pedunculopontine tegmentum, (DA) dopamine (green), (ACh) acetylcholine (yellow), (D1R) D1 dopamine receptor, (nAR) nicotinic acetylcholine receptor.

This Article

  1. Learn. Mem. 20: 580-591

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